Max Synapse
(Fig. 4A, B) [47]. In mice, and it can be expressed hM3Dq as a sign of a pattern of localization of neuronal activity (ie the pattern of activation of the promoter c-fu) in a certain period of time (ie outside Doxycycline period). First, the mice exposed to the context of the novel, and the context thousand (CtxA), to induce tagging nerve cells that were active in particular in the context of (Figure 4C, left). Second, the mice exposed to the context of another novel, CtxB, where the foot shock (US), Max Synapse and injecting the CNO (to activate hM3Dq - and thus CtxA neurons encoding) were administered (Fig. 4C, center), and thus can be associated information CtxA + CtxB with shock foot. Finally, the mice showed a freeze only when signed by injection drug Czechoslovak (pseudo CtxA) and exposure to CtxB at one time (Fig. 4C, right), but not when the injection was given CNO's exposure and CtxB alone. The result indicates that rats create a hybrid memory "synthetic" from CtxA pandemic. Using the same promoter TTA mouse line c Federal Statistics Office in combination with the viral delivery of TRE-channelrhodopsin, Liu et al. [48] showed that the optical nerve reactivate the band involved in memory